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Falciparum Malaria and Acute Renal Failure

    Author

    • Golla Vahini, Yerraguntla Shashidhar
,

Document Type : Research Article

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Abstract

Background: Aim & Objectives: To study the ABG and electrolyte disturbances in
severe malaria. To assess the prognostic significance of these parameters.
Materials and Methods: The present study is a prospective study of 50 patients above
the age of 12 yrs. The patients were selected from those who were admitted with severe
malaria in Acute Medical Care Unit, Ganndhi Hospital, Secunderabad. Patients who
came with symptoms or signs of severe malaria like coma, convulsions, hypotension,
decreased urine output, anemia, jaundice, respiratory distress are assessed. Out of them
who fit the exclusion criteria are excluded.
Results: Acidosis is commonly seen with malaria and this most often high anion gap
metabolic acidosis contributed by lactic acidosis, renal failure and other anions.
Electrolyte abnormalities are common in malaria with hyponatremia, eukalemia,
hypochloremia, hypocalcemia and hypophosphatemia being the commonest.
Conclusion: Finally, we conclude, Strong predictors of mortality include acidosis, high
anion gap, hyperlactatemia and hyperkalemia

Keywords

  • Malaria
  • hypokalemia
  • Hyponateremia
  • Jaundice
  • hypotension
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European Journal of Molecular & Clinical Medicine
Volume 9, Issue 3
February 2022
Page 914-924
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  • Article View: 108
  • PDF Download: 183

APA

Shashidhar, G. V. Y. (2022). Falciparum Malaria and Acute Renal Failure. European Journal of Molecular & Clinical Medicine, 9(3), 914-924.

MLA

Golla Vahini, Yerraguntla Shashidhar. "Falciparum Malaria and Acute Renal Failure". European Journal of Molecular & Clinical Medicine, 9, 3, 2022, 914-924.

HARVARD

Shashidhar, G. V. Y. (2022). 'Falciparum Malaria and Acute Renal Failure', European Journal of Molecular & Clinical Medicine, 9(3), pp. 914-924.

VANCOUVER

Shashidhar, G. V. Y. Falciparum Malaria and Acute Renal Failure. European Journal of Molecular & Clinical Medicine, 2022; 9(3): 914-924.

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